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发布于:2020-1-13 08:58:31  访问:34 次 回复:0 篇
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GmentsThis work was funded by Edward R. and Anne G. Lefler
Irritation in TAK-659 In Vitro Alzheimer sickness -- a brief evaluate in the primary science and clinical literature. Sutezolid Sutezolid clinical trial Hollingworth P, Harold D, Sims R, Gerrish A. Lefler Fellowship (SH), Countrywide Institutes of Overall health AG000222 (SH), Lundbeck Basis (LDO), Coins for Alzheimer‘s Study Have faith in (BS), and Anonymous (BS).Curr Opin Neurobiol. Writer manuscript; available in PMC 2017 June 21.Hong et al.PageReferences and advised readingPapers of unique interest, posted PubMed ID:https://www.ncbi.nlm.nih.gov/pubmed/23566152 within just the period of time of review, are actually highlighted as: ?of particular interest of excellent interest1. Wyss-Coray T, Rogers J. Irritation in Alzheimer illness -- a short evaluate of your fundamental science and medical literature. Chilly Spring Harb Perspect Med. 2012; two:a006346. [PubMed: 22315714] two. Bertram L, Lange C, Mullin K, Parkinson M, Hsiao M, Hogan MF, Schjeide BMM, Hooli B, DiVito J, Ionita I, et al. Genome-wide association examination reveals putative Alzheimer‘s disease susceptibility loci additionally to APOE. Am J Hum Genet. 2008; eighty three:623?32. [PubMed: 18976728] three. Lambert J-C, Heath S, Even G, Campion D, Sleegers K, Hiltunen M, Combarros O, Zelenika D, Bullido MJ, Tavernier B, et al. Genome-wide affiliation study identifies variants at CLU and CR1 associated with Alzheimer‘s disease. Nat Genet. 2009; 41:1094?099. [PubMed: 19734903] 4. Hollingworth P, Harold D, Sims R, Gerrish A. Popular variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are connected to Alzheimer‘s sickness. Character. 2011 five. Naj AC, Jun G, Beecham GW, Wang L-S, Vardarajan BN, Buros J, Gallins PJ, Buxbaum JD, Jarvik GP, Crane PK, et al. Prevalent variants at MS4A4/MS4A6E, CD2AP, CD33 and EPHA1 are affiliated with late-onset Alzheimer‘s disease. Nat Genet. 2011; 43:436?forty one. [PubMed: 21460841] 6. Guerreiro R, Wojtas A, Bras J, Carrasquillo M, Rogaeva E, Majounie E, Cruchaga C, Sassi C, Kauwe PubMed ID:https://www.ncbi.nlm.nih.gov/pubmed/28077646 JSK, Younkin S, et al. TREM2 variants in Alzheimer‘s sickness. N Engl J Med. 2013; 368:117?27. [PubMed: 23150934] seven. Jonsson T, Stefansson H, Steinberg S, Jonsdottir I, Jonsson PV, Snaedal J, Bjornsson S, Huttenlocher J, Levey AI, Lah JJ, et al. Variant of TREM2 related to the risk of Alzheimer‘s sickness. N Engl J Med. 2013; 368:107?16. [PubMed: 23150908] 8. Zhang B, Gaiteri C, Bodea L-G, Wang Z, McElwee J, Podtelezhnikov AA, Zhang C, Xie T, Tran L, Dobrin R, et al. Integrated methods strategy identifies genetic nodes and networks in late-onset Alzheimer‘s disease. Cell. 2013; 153:707?twenty. [PubMed: 23622250] nine. Lambert J-C, Ibrahim-Verbaas CA, Harold D, Naj AC, Sims R, Bellenguez C, Jun G, DeStefano AL, Bis JC, Beecham GW, et al. Meta-analysis of 74,046 men and women identifies eleven new susceptibility loci for Alzheimer‘s disease. Nat Genet. 2013; forty five:1452?458. [PubMed: 24162737] 10. Meyer-Luehmann M, Spires-Jones TL, Prada C, Garcia-Alloza M, De Calignon A, Rozkalne A, Koenigsknecht-Talboo J, Holtzman DM, Bacskai BJ, Hyman BT. Swift visual appearance and local toxicity of amyloid- plaques in a mouse model of Alzheimer‘s sickness. Character. 2008; 451:720?724.
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